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SAC regulates glutathione metabolism via the Keap1/Nrf2/GPX4 signaling pathway to inhibit ferroptosis and alleviate ulcerative colitis | Scientific Reports
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[ 127 ] Therefore, there are at least two mechanisms for BACH1 deficiency to alleviate atherosclerotic lesion formation: firstly, it can have an anti‐inflammatory impact by suppressing YAP activation in ECs, and secondly, it can produce anti‐oxidative effects dependent on HO‐1
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