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On these grounds, Zhang and colleagues hypothesized that H 2 S induced VAMP3 degradation through Hrd1 S-sulfhydration and prevented CD36 translocation, thereby alleviating lipid toxicity in the heart tissues of db/db diabetic mice (Yu et al., 2020)

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Among the pathways discussed, lipid metabolic rewiring currently appears the most promising targetable axis to overcome TMZ resistance, given consistent in vivo sensitization data and the fact that several lipid-directed approaches have already entered early-phase clinical evaluation