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They control Toll-like receptor (TLR) and G-protein coupled receptor (GPR) pathways and stimulate anti-inflammatory regulatory agents like A20, Bcl-3, and MKP-1, inhibiting lipopolysaccharide (LPS)-stimulated TLR4 activation [226]
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According to pyramid of medical evidence, the majority of clinical study summarized in this review represent the highest levels of clinical evidence including randomized clinical trials (RCTs) and network meta-analysis (NMA)
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