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To investigate the effect of GPx4 inactivation on hepatic cell viability, we knocked down GPx4 using siRNAs or shRNA lentivirus in AML12 cells, which are derived from normal mouse hepatocytes (Fig
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The HopQ-CEACAM interaction also enables the translocation of the virulence factor cytotoxin-associated gene A (CagA) into host cells (Javaheri et al., 2016
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