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1.4.3 Limitations of benign and VUS classifications Ultimately, WES is not a genetic oracle, and in contrast to Mendelian diseases– which often result from single-gene coding variants (Botstein and Risch, 2003), several complex traits could be driven by noncoding variants that presumably affect gene regulation

R-Lipoic Acid

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Molecular Mechanisms, In Vitro, and In Vivo Studies Divergent mechanisms such as competition for transporters and binding sites (DMT1, ZIP8/14), MT induction via MTF-1, Nrf2-driven antioxidant support, maintenance of epithelial barriers, and immune modulation are involved in mitigating HMs toxicity [127,128,129]