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The homological resemblance between thyroxine (TH) and particular TH-disrupting chemicals (TH-EDCs), such as PCB metabolites, dioxin (PCDD), and brominated flame retardants, results in binding to the high-affinity TH transport protein, transthyretin TTR, thereby obstructing T4-TTR binding (Grimm et al., 2013)
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