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Final results of a multicenter phase II study of the purine nucleoside phosphorylase (PNP) inhibitor forodesine in patients with advanced cutaneous T-cell lymphomas (CTCL) (Mycosis fungoides and Sézary syndrome)
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The inflammatory response activates the PKM2 dimer, leading to its translocation to the nucleus, where it phosphorylates and activates transcription factors such as Nrf2, STAT3, and NF-κB, thereby regulating gene expression
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