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Exogenetic miR-144-3p expression compromised the malignant biological properties of GBM cells independent of PTEN and resulted in enhancement of radiation and temozolomide (TMZ) sensitivity by binding to c-Met and thus impairing the activity of downstream signaling (Lan et al., 2015)
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This increase in mucin and proenzymes provides sufficient materials for the rapid turnover of proteins in the cells of the intestinal epithelium, particularly during aflatoxicosis
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