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Notably, mutations in PROP1 (Prop1df/df, Ames dwarf mice) and POU1F1 (Pou1f1dw/dw, Snell dwarf mice) disrupt anterior pituitary development, leading to deficiencies in GH, thyroid-stimulating hormone (TSH), and prolactin (PRL), significantly extending lifespan (Junnila et al., 2013)
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