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The active form of VD (calcitriol) inhibits the production of proinflammatory cytokines, such as TNF-α, IL-6, and IL-17, while supporting the function of Tregs, which help control the body’s autoimmune response
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The 2020 systematic review by Galeano and colleagues across 34 eligible studies confirmed the mechanism: PDRN promotes physiological tissue repair through salvage-pathway nucleotide recycling and A2A receptor activation [32]
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