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GHK-Cu is reported to influence a large number of genes and to accelerate epithelialization in wound models, but these findings describe biological activity, not a validated human safety margin
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Deficiency in the level of activity of G6PDH is the basis of favism, primaquine (an antimalarial drug) sensitivity, and several other drug-sensitive hemolytic anemias, anemia and jaundice in the newborn, and chronic nonspherocytic hemolytic anemia
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