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298 Under excessive ROS levels, ATM triggers downstream sites such as p53 and checkpoint kinase 2 (CHK2) for activation even when the DNA damage response (DDR) is absent, indicating that the activation pathway has no impact on the role of ATM at a certain level, whereas ATM has many other functions independent of the DDR 297 (Table 1, Fig

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ARHGEF2 counteracts ARHGAP15 to regulate autophagy and centrosome numbers To understand the centrosome signalling mechanism that could trigger a rise in RAC1-GTP and activate autophagy, we sought to identify a guanine-nucleotide exchange-factor (GEF) counteracting ARHGAP15