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3 Dual targeting for optimal treatment of autoimmune diseases Early in the disease course, CD19-based therapy may prevent autoreactive plasma cell accumulation, but later, memory plasma cells may accumulate and lead to persistent autoantibody production despite B-cell depletion (32, 33)

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Locus outer membrane-bound non-catalytic SGBPs plays essential roles in recruiting and capturing high MW barley- β- glucan, and the SusD allows mixed-linkage β- (1,3)/β-(1,4) glucan-oligosaccharides to enter in periplasmic space in concert with cognate TonB-dependent transporters (TBDTs) as shown in Figure 2