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Subsequently, regardless of the mechanism involved, in mitochondrial DNA damage or in the direct involvement of prooxidant factors in cellular mechanisms, the cellular response to stress will produce an overexpression of proinflammatory genes with increasing levels of prooxidant factors (Liguori et al., 2018)
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[DOI] [PMC free article] [PubMed] [Google Scholar] 17.De Duve C., Baudhuin P
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